Classical Double Copy and Higher-Spin Fields.

Kerr-Schild double copy is shown to extend naturally to all free symmetric gauge fields propagating on (A)dS in any dimension. Similarly to the standard lower-spin case, the higher-spin multicopy comes along with the zeroth, single, and double copies. The masslike term of the Fronsdal spin s field equations fixed by gauge symmetry and the mass of the zeroth copy both appear to be remarkably fine-tuned to fit the multicopy pattern forming a spectrum organized by higher-spin symmetry.

[Ontogenetic characteristics of patients with erectile dysfunction with different types of aging.].

Age-related erectile dysfunction (ED) can be formed during ontogenesis in accordance with various mechanisms of the pathogenesis of aging in conjunction with a particular cluster of diseases. The aim of the work is to study ontogenetic features of ED patients in late ontogeny of various types of aging. 65 men over 45 with an explicit form of ED were examined.

Zebra Tail Amplification: Accelerated Detection of Apoptotic Blunt-Ended DNA Breaks by In Situ Ligation.

In situ ligation (ISL) is a simple and specific technique for apoptosis labeling in tissue sections. In its most economical version ISL uses ordinary PCR-labeled DNA fragments as probes. In tissue sections these makeshift probes are ligated to apoptotic DNA breaks by T4 DNA ligase.

Quick Detection of DNase II-Type Breaks in Formalin-Fixed Tissue Sections.

Blunt-ended DNase II-type breaks with 5' hydroxyls are generated in phagocytic cells of any lineage during digestion of the engulfed DNA. These breaks indicate the ongoing active phagocytic reaction. They are produced by the acid deoxyribonuclease-DNase II which is the primary endonuclease responsible for DNA degradation after its engulfment.

Express FRET Labeling and Analysis of Phagocytic Clearance.

Lysosomal DNase II in phagocytic digestion produces DNA ends with 3'PO/5'OH, which differ from those created in apoptotic DNA fragmentation, and can be used to label phagocytic clearance of cell death. Here, we describe the use of these specific DNA ends as selective markers of phagocytic reaction in cell suspensions. The approach does not require cell fixation.