Increased intracranial arterial resistance in patients with type 2 diabetes mellitus.

Background: The majority of the studies investigating risk factors for stroke have focused on the atherosclerosis of extracranial carotid arteries. Risk factors for the involvement of intracranial arteries in patients with stroke have not been widely investigated so far. The pulsatility index reflects the vascular resistance of intracranial arteries and could therefore be used as an estimate of the severity of vascular damage.

[Changes in circulation of the blood in cerebral arteries in relation to age].

The authors examined 12 people under 40 years and 12 above 40 years, mostly healthy volunteers. They investigated the rate of blood flow in the median cerebral artery after compression of the homolateral and contralateral internal carotid. They found that in more advanced age the rate of flow in the mentioned arteries is significantly reduced.

[Cerebral circulation using transcranial and extracranial doppler ultrasonography in a patient with vertigo and ischemia in the area of the brain stem].

The authors examined 32 patients with vertigo, 17 patients with focal ischaemia of the brain stem and 24 patients without signs of affections of the central nervous system by transcranial and extracranial Doppler ultrasonography. They measured the flow velocity in the basilar artery (BA), in the intracranial portion of the vertebral artery (VA) and in the extracranial portion of the internal carotid artery (ICA). In patients with vertigo they investigated functional and organic changes in the area of the cervical spine.

[Glucose-6-phosphate dehydrogenase and catalase in the erythrocytes of patients with cerebral ischemia attacks].

The authors assessed the activity of glucose-6-phosphate dehydrogenase and catalase in 73 patients with ischaemic cerebral attacks. They compared the results with a control group of 23 patients with vertebrogenic diseases. They recorded a mild decrease of activity of the above enzymes and draw attention to possible oxidation damage under conditions of cerebral hypoxia.