Validation of the Mitochondrial Delivery of Vitamin B to Enhance ATP Production Using SH-SY5Y Cells, a Model Neuroblast.

Large amounts of ATP are produced in mitochondria especially in the brain and heart, where energy consumption is high compared with other organs. Thus, a decrease in ATP production in such organs could be a cause of many diseases such as neurodegenerative diseases and heart disease. Based on thus assumption, increasing intracellular ATP production in such organs could be a therapeutic strategy.