Publications by authors named "T Z Amer"

Neutron and gamma-ray shielding design for a 30Ci (1.11TBq) Am-Be irradiation facility is studied using MCNP5 Monte Carlo simulation code. The study focuses on the optimization of the shielding layers of the previously planned neutron irradiation facility.

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This study aims to demonstrate the behaviors of a two degree-of-freedom (DOF) dynamical system consisting of attached mass to a nonlinear damped harmonic spring pendulum with a piezoelectric device. Such a system is influenced by a parametric excitation force on the direction of the spring's elongation and an operating moment at the supported point. A negative-velocity-feedback (NVF) controller is inserted into the main system to reduce the undesired vibrations that affect the system's efficiency, especially at the resonance state.

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Genetic screens in have long been used to identify genes found in a variety of developmental, cellular, and behavioral processes. Here we describe the characterization and mapping of a mutation identified in a conditional screen for genetic regulators of cell growth and cell division. Within a Flp/FRT system, mutant results in a reduction of mutant tissue and a rough eye phenotype.

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The primary objective of this study is to explore the spatial rotary movements of a symmetrically charged rigid body (RB) that is rotating around a fixed point, akin to Lagrange's scenario as a novel scenario where its center of mass experiences a slight displacement from the symmetry dynamic axis. The body's movement is presumed to be affected by a gyrostatic moment and a force from an electromagnetic field, attributed to the presence of a located point charge on this axis. The regulating equations of motion that are pertaining to the equations Euler-Poisson are solved through the utilization of Poincaré's small parameter method along with its adaptations when the scenario of irrational frequencies is considered.

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Cellular senescence is a response to many stressful insults. DNA damage is a consistent feature of senescent cells, but in many cases its source remains unknown. Here, we identify the cellular endonuclease caspase-activated DNase (CAD) as a critical factor in the initiation of senescence.

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