[Alkaline blood serum proteins in patients with the initial manifestations of cerebral arteriosclerosis and the late sequelae of mild closed craniocerebral trauma (comparative data)].

Alkaline proteins play their pathogenetic part in the course of cerebral atherosclerosis the development of which is accompanied by decline in their content. It has been ascertained that in patients with early cerebral atherosclerosis and remote sequelae of mild closed craniocerebral injury (RS MCCCI) the content of alkaline proteins is below the established norm. The absence of significant differences between the patients presenting with early manifestation of cerebral circulatory insufficiency and those with RS MCCCI may suggest similarity of mechanisms of development of vascular disorders of traumatic and atherosclerotic genesis.

[The place and significance of the autonomic dystonia syndrome in the clinico-pathophysiological structure of the late sequelae of mild closed craniocerebral trauma].

Analysis of the incidence, clinico-pathophysiological structure and dynamics of vegetovascular disorders in subjects with a history of mild closed craniocerebral injuries has demonstrated that in the majority of them, even practically healthy, functional insufficiency of vegetovascular functions is seen for many years after injury. Clinically, it manifests under the influence of diverse harmful exo- and endogenous factors, undergoes circadian changes, is altered during magnetic storms, in the course of traumatic disease and nonmedicamentous correction (by methods of adaptive bioregulation according to heart rhythm parameters, craniocerebral hypothermia, etc). It has been shown that initially transitory, reversible vegetovascular disturbances, provided they were not initially removed, transform with years to more stable vegetotrophic disorders and become risk factor of cerebrovascular diseases in the given group.