Publications by authors named "T Meulia"

Maize lethal necrosis is a destructive virus disease of maize caused by maize chlorotic mottle virus (MCMV) in combination with a virus in the family Potyviridae. Emergence of MLN is typically associated with the introduction of MCMV or its vectors and understanding its spread through seed is critical for disease management. Previous studies suggest that although MCMV is detected on seed, the seed transmission rate of this virus is low.

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Article Synopsis
  • Almonds are a valuable nut crop primarily produced in the U.S., with 'Nonpareil' being the most popular cultivar, but until now, there was no whole-genome assembly available for it.
  • Researchers generated a comprehensive genome sequence for the 'Nonpareil' almond using advanced sequencing technologies, revealing over 49,000 possible genes, with a significant portion linked to biological functions.
  • The study also explored the methylation patterns across different almond tissues, showing variations that may affect traits like self-compatibility and bud dormancy.
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Entodinium caudatum is an anaerobic binucleated ciliate representing the most dominant protozoal species in the rumen. However, its biological features are largely unknown due to the inability to establish an axenic culture. In this study, we primally sequenced its macronucleus (MAC) genome to aid the understanding of its metabolism, physiology, ecology.

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Rubber dandelion ( Rodin; TK) has received attention for its natural rubber content as a strategic biomaterial, and a promising, sustainable, and renewable alternative to synthetic rubber from fossil carbon sources. Extensive research on the domestication and rubber content of TK has demonstrated TK's potential in industrial applications as a relevant natural rubber and latex-producing alternative crop. However, many aspects of its biology have been neglected in published studies.

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We recently reported that the p28 auxiliary replication protein encoded by turnip crinkle virus (TCV) is also responsible for eliciting superinfection exclusion (SIE) against superinfecting TCV. However, it remains unresolved whether the replication function of p28 could be separated from its ability to elicit SIE. Here, we report the identification of two single amino acid mutations that decouple these two functions.

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