Shallow versus deep genetic causes.

We argue that Madole & Harden's distinction between shallow versus deep genetic causes can bring some clarity to causal claims arising from genome-wide association studies (GWASs). However, the authors argue that GWAS only finds shallow genetic causes, making GWAS commensurate with the environmental studies they hope to supplant. We also assess whether their distinction applies best to explanations or causes.

Changes in heritability: Unpredictable and of limited use.

We argue that heritability estimates cannot be used to make informed judgments about the populations from which they are drawn. Furthermore, predicting changes in heritability from population changes is likely impossible, and of limited value. We add that the attempt to separate human environments into cultural and non-cultural components does not advance our understanding of the environmental multiplier effect.

Evolutionary perspectives on human aggression: introduction to the special issue.

The papers in this volume present varying approaches to human aggression, each from an evolutionary perspective. The evolutionary studies of aggression collected here all pursue aspects of patterns of response to environmental circumstances and consider explicitly how those circumstances shape the costs and benefits of behaving aggressively. All the authors understand various aspects of aggression as evolved adaptations but none believe that this implies we are doomed to continued violence, but rather that variation in aggression has evolutionary roots.

Alternative splicing, the gene concept, and evolution.

Alternative splicing allows for the production of many gene products from a single coding sequence. I introduce the concept of alternative splicing via some examples. I then discuss some current hypotheses about the explanatory role of alternative splicing, including the claim that splicing is a significant contributor to the difference in complexity between the human genome and proteosome.