Publications by authors named "Stephany Angelia Tumewu"
Background: Cucumber Fusarium wilt (CFW), triggered by Fusarium oxysporum f. sp. cucumerinum, leads to substantial yield reductions in global cucumber (Cucumis sativus L.
View Article and Find Full Text PDFPseudomonas syringae pv. tabaci 6605 (Pta6605) is a foliar plant pathogen that causes wildfire disease on tobacco plants. It requires chemotaxis to enter plants and establish infection.
View Article and Find Full Text PDFChemotaxis is crucial for Pseudomonas syringae pv. tabaci (Pta) 6605 to evoke disease in tobacco plants. Pta6605 harbors more than fifty genes for methyl-accepting chemotaxis proteins (mcp), but almost all are functionally uncharacterized.
View Article and Find Full Text PDFArticle Synopsis
- Pseudomonas syringae pv. tabaci 6605 (Pta6605) is a highly mobile bacterium responsible for wildfire disease in tobacco plants and contains multiple chemotaxis gene clusters.
- Research involved creating deletion mutants of key genes cheA and cheY to assess their roles in the bacterium's movement and ability to cause disease.
- Results revealed that mutants lacking genes from cluster II (cheA2 and cheY2) could not swarm or exhibit chemotaxis and did not cause disease symptoms, while mutations in cluster I had a less significant impact on chemotaxis and disease-causing ability.
Article Synopsis
- GABA (γ-Aminobutyric acid) acts as a signaling molecule in plants, especially during stress and interactions with harmful bacteria, but its mechanisms are not fully understood.
- Researchers identified a GABA receptor named McpG in the plant-pathogenic bacterium Pseudomonas syringae pv. tabaci 6605 (Pta6605) and created a mutant lacking this receptor to study its role in GABA chemotaxis.
- The findings showed that while Pta6605 normally responds to GABA, the ΔmcpG mutant lost this attraction and was less effective in causing disease in tobacco plants, indicating that GABA sensing is crucial for the bacterium's interaction with its host.