Publications by authors named "Stephanie Ashton"

Intrusive memories can be downregulated using intentional memory control, as measured via the Think/No-Think paradigm. In this task, participants retrieve or suppress memories in response to an associated reminder cue. After each suppression trial, participants rate whether the association intruded into awareness.

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Negative outlooks of our future may foster unwanted and intrusive thoughts. To some extent, individuals have control over their ability to suppress intrusions and downregulate their frequency. Acute stress impairs intentional suppression, leading to an increased frequency of intrusions.

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Article Synopsis
  • Women experiencing physical intimate partner violence (IPV) are at a heightened risk for brain injuries (BIs), but many shelter staff lack awareness of this link.
  • A new online training module was developed to educate shelter staff about IPV-caused BIs, and a study found significant improvements in knowledge and advocacy skills among participants after completing it.
  • Feedback from participants indicated increased knowledge, mindfulness, and a desire to recommend the training to colleagues, suggesting that the training effectively enhances care for women with IPV-related BIs.
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Instrumental learning is controlled by two distinct parallel systems: goal-directed (action-outcome) and habitual (stimulus-response) processes. Seminal research by Schwabe and Wolf (2009, 2010) has demonstrated that stress renders behavior more habitual by decreasing goal-directed control. More recent studies yielded equivocal evidence for a stress-induced shift towards habitual responding, yet these studies used different paradigms to evaluate instrumental learning or used different stressors.

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To some extent, we can shape our recollections by intentionally remembering certain experiences while trying to forget others, for example, by intentional suppression. Acute stress impairs suppression-induced forgetting of memories. It is unclear, however, whether these deficits are a direct consequence of the acute stress-induced cortisol response.

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