Researchers identified the roles of Methoprene-tolerant (Met) and Krüppel-homolog 1 (Kr-h1) in reproductive processes of the insect Liposcelis entomophila, focusing on vitellogenesis and ovarian development.
Treatment with juvenile hormone (JH III) increased the levels of key proteins involved in reproduction, while silencing Met and Kr-h1 reduced these proteins, affecting egg production and lowering fertility.
Additionally, disrupting these genes caused higher mortality rates in the insect, suggesting that targeting JH signaling could be a viable pest control method.