3'--β-d-glucopyranosyl-α,4,2',4',6'-pentahydroxy-dihydrochalcone, from Bark of Prevents Diabetic Nephropathy via Inhibiting Protein Glycation in STZ-Nicotinamide Induced Diabetic Mice.

Previous studies have shown that accumulation of advanced glycation end products (AGEs) can be the cause of diabetic nephropathy (DN) in diabetic patients. Dihydrochalcone 3'--β-d-glucopyranosyl α,4,2',4',6'-pentahydroxy⁻dihydrochalcone () is a powerful antiglycation compound previously isolated from . The aim was to investigate whether () was able to protect against diabetic nephropathy in streptozotocin (STZ)-induced diabetic mice, which displayed renal dysfunction markers such as body weight, creatinine, uric acid, serum urea, total urinary protein, and urea nitrogen in the blood (BUN).

A comparative study of physical and chemical processes for removal of biomass in biofilters.

After 6 months of operation a long-term biofilter was stopped for two weeks and then it was started up again for a second experimental period of almost 1.3 years, with high toluene loads and submitted to several physical and chemical treatments in order to remove excess biomass that could affect the reactor's performance due to clogging, whose main effect is a high pressure drop. Elimination capacity and removal efficiency were determined after each treatment.