Publications by authors named "Sarah Rickard"

Objectives: Study objectives were to explore nurses' perceptions of self-care, co-worker, and leader caring within healthcare work environments and assess reliability of 3 Watson Caritas Scores.

Background: Assessing caring in an organization where Watson's Theory of Human Caring guides nursing practice offers insight into the professional practice environment.

Method: This study reports quantitative data from mixed-methods descriptive, cross-sectional survey of 1307 RNs at a large healthcare system.

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The Clean Water Act requires states to develop methods for assessing water quality. Assessment methods serve as decision-making procedures for including waterbodies on the Section 303(d) List of Impaired Waters. We used 17 years of ambient water quality data to explore statistical analyses for assessment methods that represent New York's waterbodies.

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Background And Aims: Concerns have arisen regarding patient access and delivery of acute stroke care during the COVID-19 pandemic. We investigated key population level events on activity of the three hyperacute stroke units (HASUs) within Greater Manchester and East Cheshire (GM & EC), whilst adjusting for environmental factors.

Methods: Weekly stroke admission & discharge counts in the three HASUs were collected locally from Emergency Department (ED) data and Sentinel Stroke National Audit Programme core dataset prior to, and during the emergence of the COVID-19 pandemic (Jan 2020 to May 2020).

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Article Synopsis
  • Angelman Syndrome (AS) is a neurobehavioral disorder characterized by symptoms like mental retardation, absent speech, and seizures, with specific genetic mutations confirming its diagnosis in many patients.
  • In a study of 45 patients with AS who lacked clear genetic abnormalities, higher rates of UBE3A mutations were found in familial cases compared to sporadic cases, suggesting a different underlying mechanism may exist for sporadic AS.
  • Although MECP2 mutations can sometimes present features similar to AS, they do not explain the majority of sporadic AS cases, as researchers found no MECP2 mutations in most sporadic cases they tested.
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Albright hereditary osteodystrophy (AHO) is caused by heterozygous deactivating GNAS1 mutations. There is a parent-of-origin effect. Maternally derived mutations are usually associated with resistance to parathyroid hormone termed "pseudohypoparathyroidism type Ia.

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