Transcranial electrostimulation activates reparative regeneration and the insulin-producing function of pancreatic B-cells in alloxan diabetes in rats.

Studies on rats with experimental diabetes induced by administration of alloxan showed that transcranial electrostimulation of endorphinergic brain structures stimulates the regeneration of damaged beta-cells in pancreatic islets of Langerhans. This was identified on pancreatic sections stained with hematoxylin and eosin. De novo formation of small islets was noted, as evidenced by their regeneration from progenitor cells.

[Transcranial electrical stimulation activates the reparative regeneration and insulin-producing function of beta-cells in alloxan-induced diabetic rats].

In alloxan-induced diabetic rats, it was demonstrated that transcranial electrical stimulation of the brain endorphinergic structures activated the reparative regeneration of the damaged beta-cells of the Langerhans pancreatic islets. This was estimated on the histological sections of pancreas with hematoxylin-eosin staining. Several small newborn islets were found to originate from pancreatic progenitor cells.

[Bladder foreign body in a 4-year-old girl].

The specific feature of this case presented is the prolonged clinical manifestations of cystitis despite long-term and diversified antibacterial therapy. Repeated ultrasonography was justifiable in this cases since it could reveal a cause of protracted cystitis. Thus, in the young child long-term dysuria and urinary infection may be caused by a bladder foreign body.