It is proposed that the raised serum glutamic pyruvic transaminase activity (SGPT) in viral hepatitis reflects an adaptive increase in glutamic pyruvic transminase in muscle for the increased transamination of alanine for post-absorptive glucose homeostasis. However, in a disease where the SGPT is elevated for four to six weeks, muscle cannot be an indefinite source of alanine unless it could, concurrently, resynthesize alanine. Since the two substrates of muscle are glucose and fat, fasting blood sugars, serum triglycerides and SGPT were estimated weekly, for one month, in 12 children with viral hepatitis.
View Article and Find Full Text PDFAccidental subarachnoid injection of gallamine and its management is reported. Methods of identification of the drug in the c.s.
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