A 128-channel receive array with enhanced signal-to-noise ratio performance for 10.5T brain imaging.

Purpose: To develop and characterize a 128-channel head array for brain imaging at 10.5 T, evaluate signal-to-noise ratio (SNR) relative to ultimate intrinsic SNR (uiSNR) and lower field strengths, and demonstrate human brain anatomical and functional imaging with this unique magnetic field and high-channel-count array.

Methods: The coil consists of a 16-channel self-decoupled loop transmit/receive (16Tx/Rx) array with a 112-loop receive-only (Rx) insert.

Establishment of a Biomarker-Directed Clinical Endpoint Model for Early-Stage Parkinson's Disease Patients.

Parkinson's Disease (PD) is a neurodegenerative disorder characterized by dopaminergic cell death in the substantia nigra. While the interplay between dopamine loss and symptoms is well-recognized, a respective quantitative link has yet to be established. The objective was to establish a biomarker-directed clinical endpoint model for early-stage PD patients.

Proceedings of the 12th annual deep brain stimulation think tank: cutting edge technology meets novel applications.

The Deep Brain Stimulation (DBS) Think Tank XII was held on August 21st to 23rd. This year we showcased groundbreaking advancements in neuromodulation technology, focusing heavily on the novel uses of existing technology as well as next-generation technology. Our keynote speaker shared the vision of using neuro artificial intelligence to predict depression using brain electrophysiology.

Accelerometer-based estimation of respiratory rate using principal component analysis and autocorrelation.

Respiratory rate (RR) is an important vital sign but is often neglected. Multiple technologies exist for RR monitoring but are either expensive or impractical. Tri-axial accelerometry represents a minimally intrusive solution for continuous RR monitoring, however, the method has not been validated in a wide RR range.

[Agonist-antagonist myoneural interface (AMI) : Innovative treatment option for lower limb amputees?].

The agonist-antagonist myoneural interface (AMI) is an innovative approach to restoring proprioception and achieving more intuitive motor control following limb loss. This cutting-edge technique replicates the natural biomechanical relationship between agonist and antagonist muscles, enabling bidirectional communication between a prosthesis and the user's peripheral nervous system. Through the transposition of neurovascularly pedicled agonist-antagonist muscle pairs, which are reconnected via an adapted tendon suture and positioned within a gliding mechanism, AMI generates proprioceptive feedback during movement.