Loss of short-latency afferent inhibition and emergence of afferent facilitation following neuromuscular electrical stimulation.

Neuromuscular electrical stimulation (NMES) increases the excitability of corticospinal (CS) pathways by altering circuits in motor cortex (M1). How NMES affects circuits interposed between the ascending afferent volley and descending CS pathways is not known. Presently, we hypothesized that short-latency afferent inhibition (SAI) would be reduced and afferent facilitation (AF) enhanced when NMES increased CS excitability.

"Metallic" and "insulating" behavior of the two-dimensional electron gas on a vicinal surface of Si MOSFET'S.

The resistance R of the 2DEG on the vicinal Si surface shows unusual behavior which is very different from that in Si (100) MOSFET's studied earlier. The low-temperature crossover from dR/dT<0 ("insulator") to dR/dT>0 ("metal") occurs at a low resistance of R(c)square approximately 0.04xh/e2.