Publications by authors named "S Lamquet"

Article Synopsis
  • * It has not been widely used in clinical settings but shows promise as a long-term treatment for focal drug-resistant epilepsy, especially when the seizures originate in the motor cortex.
  • * A case study of a 49-year-old woman with severe focal motor seizures demonstrated that CSCS led to significant improvement, allowing her to walk independently for the first time in years.
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Autoimmune encephalitis refers to a group of disorders characterised by a non-infectious encephalitis, often with prominent seizures and surface neuronal autoantibodies. AE is an important cause of new-onset refractory status epilepticus in humans and is frequently responsive to immunotherapies including corticosteroids, plasma exchange, intravenous immunoglobulin G and rituximab. Recent research suggests that parallel autoantibodies can be detected in non-human mammalian species.

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Cerebral abscess formation is a serious and life-threatening clinical entity, secondary to contiguous spread, hematogenous dissemination or direct inoculation. We present the case of a 61-year-old woman with a recent diagnosis of a locally advanced squamous cell carcinoma of the esophagus who was diagnosed with a brainstem abscess. In literature we only found three cases reporting cerebral abscess formation in patients with esophageal carcinoma.

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Introduction: The radial head has an ellipsoid shape so that a longest and a shortest axis can be defined. The aim of this study is to evaluate the position of the longest axis of the radial head (LARH) in relation to proximal radioulnar joint (PRUJ) and to the forearm in neutral position using 3D computed tomography (CT).

Materials And Methods: 3D CT reconstructions of the distal humerus, the radius and the ulna of 27 healthy volunteers (average age 27.

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A 60-year-old man is presented with progressive involuntary muscle movements and neuropsychiatric symptoms who developed a variety of additional complaints over 2 years. Brain imaging revealed bilateral basal ganglia calcifications suggesting primary familial brain calcification. Analysis of the gene revealed a missense mutation (c.

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