and in Turkeys in Poland.

The pathogenic mycoplasmas are among the bacteria causing significant losses in the poultry industry worldwide. (MG) and (MS) are economically important pathogens causing chronic respiratory disease, decreased growth, egg production and hatchability rates, and significant downgrading of carcasses. Effective diagnosis of infection with these species in poultry is highly requisite considering their two routes of spreading-horizontal and vertical.

Insufficient generation of Th17 cells in IL-23p19-deficient BALB/c mice protects against progressive cutaneous leishmaniasis.

Healing of leishmaniasis-a parasitic skin disease-is associated with high levels of secreted interferon (IFN)γ and IL-12 in resistant C57BL/6 mice and humans. Susceptible BALB/c mice predominantly react with a Th17/Th2/Treg-related immune response and finally succumb to infection. Previously, we showed that BALB/c IL-17A mice are protected against Leishmania (L.

Dynamical-systems analysis and unstable periodic orbits in reacting flows behind symmetric bluff bodies.

Dynamical systems analysis is performed for reacting flows stabilized behind four symmetric bluff bodies to determine the effects of shape on the nature of flame stability, acoustic coupling, and vortex shedding. The task requires separation of regular, repeatable aspects of the flow from experimental noise and highly irregular, nonrepeatable small-scale structures caused primarily by viscous-mediated energy cascading. The experimental systems are invariant under a reflection, and symmetric vortex shedding is observed throughout the parameter range.

A role for leukocyte-derived IL-1RA in DC homeostasis revealed by increased susceptibility of IL-1RA-deficient mice to cutaneous leishmaniasis.

Dendritic cell (DC)-derived IL-1α/β plays a critical role in the induction of T helper type 1 (Th1)-dependent immunity against Leishmania. DCs from susceptible BALB/c mice produce less IL-1α/β when compared with resistant C57BL/6 mice, contributing to aberrant Th2 development and ultimate death of infected mice. We have extended our studies of the role of IL-1 in leishmaniasis using IL-1RA(-/-) BALB/c mice that are characterized by upregulated IL-1 receptor signaling.