[Cytochemical study of the natural resistance of the body in Trichophyton rubrum-induced mycosis (tinea)].

Clinical-cytochemical examination of 27 patients with tinea has permitted estimating nonspecific immune status of an organism. The NBT-test is shown to be highly informative and active. Differences in metabolic activity of phagocytizing subpopulations of peripheral blood are revealed.

[Oxygen-dependent phagocytosis in patients with tinea pedis during treatment].

Clinico-cytochemical (with the use of NBT test) studies of 27 patients with T. rubrum-induced trichophytosis of the soles has demonstrated a functional imbalance of the peripheral blood phagocytizing cells. Reduced neutrophil activity is associated with a compensatory elevation of mononuclear fungicidal potential.

[The nonspecific correction of phagocytosis in patients with foot mycoses].

A clinico-cytochemical examination of 47 patients with mycosis pedis is presented. The author established that these patients showed an inhibition of the phagocytic activity of neutrophils due to imbalance of acid and alkaline phosphatases. It is considered justified to use diuciphon as a means of unspecific immunocorrection.

[Oxygen-dependent indicators of phagocytosis in an infection caused by Trychophyton rubrum].

A clinico-cytochemical investigation of 27 patients with rubrophytia revealed a suppression of phagocytic activity of neutrophils conjugated with inhibition of the nitroblue tetrazolium test. It was confirmed that inclusion into the therapeutic complex of chlorophyllipt allows to level the intraleucocytic metabolic disbalance, to achieve enhanced regression of clinical manifestations of mycosis.

[The glycolytic status of peripheral blood phagocytes and its therapeutic correction in patients with foot mycoses].

Changed glycogen levels in the neutrophils and monocytes is a pathogenetic component contributing to the development of mycoses of the soles and a characteristic feature of nonspecific immune reactivity. The neutrophilic phagocytic activity is depressed in such patients, and the monocytes are functionally inert. Furazolidonum, potassium orotate, and riboflavin are effective stimulants of the cell-mediated defense in such patients.