Reproducible Quantitative Trait Loci for Resistance to Soft Rot Caused by Derived from the Wild Potato (PI 458355) Are Located on Chromosomes 1, 3, and 5.

The potato wild relative is a breeder-friendly source of genetic resistance to soft rot. Our objectives were to (i) identify loci associated with soft rot resistance in germplasm and (ii) develop bi-parental populations in a self-compatible genetic background to recover segregating F progenies, construct a linkage map, and identify quantitative trait loci (QTLs). Under objective (i), tubers from 103 genotypes from the United States Potato Genebank were inoculated with a high virulence strain of , and lesion size was measured after a 24-h incubation period at 30°C.

Selection strategies to introgress water deficit tolerance derived from accession LA1141 into cultivated tomato.

Crop wild relatives have been used as a source of genetic diversity for over one hundred years. The wild tomato relative accession LA1141 demonstrates the ability to tolerate deficit irrigation, making it a potential resource for crop improvement. Accessing traits from LA1141 through introgression may improve the response of cultivated tomatoes grown in water-limited environments.

Novel Isolates Alleviate Water Deficit Stress in Susceptible Tomato Genotypes.

Symbiotic fungi in the genus can induce abiotic stress tolerance in crops. The beneficial effects of on water deficit stress are poorly understood and may be isolate-specific. Our objective was to evaluate a collection of Nepalese isolates and their efficacy to improve tomato () growth under water deficit.

-derived purple tomato fruit color is conferred by novel alleles of the and loci.

One hypothesis for the origin of endemic species of tomato on the Galápagos islands postulates a hybridization of and . accession LA1141 has purple fruit pigmentation, previously described in green-fruited wild tomatoes such as . or .

Effects of experimental strabismus on the architecture of macaque monkey striate cortex.

Strabismus, a misalignment of the eyes, results in a loss of binocular visual function in humans. The effects are similar in monkeys, where a loss of binocular convergence onto single cortical neurons is always found. Changes in the anatomical organization of primary visual cortex (V1) may be associated with these physiological deficits, yet few have been reported.