Face Presentation Attack Detection Using Deep Background Subtraction.

Currently, face recognition technology is the most widely used method for verifying an individual's identity. Nevertheless, it has increased in popularity, raising concerns about face presentation attacks, in which a photo or video of an authorized person's face is used to obtain access to services. Based on a combination of background subtraction (BS) and convolutional neural network(s) (CNN), as well as an ensemble of classifiers, we propose an efficient and more robust face presentation attack detection algorithm.

Spatiotemporal CNN with Pyramid Bottleneck Blocks: Application to eye blinking detection.

Eye blink detection is a challenging problem that many researchers are working on because it has the potential to solve many facial analysis tasks, such as face anti-spoofing, driver drowsiness detection, and some health disorders. There have been few attempts to detect blinking in the wild scenario, while most of the work has been done under controlled conditions. Moreover, current learning approaches are designed to process sequences that contain only a single blink ignoring the case of the presence of multiple eye blinks.

Molecular detection of the B1 gene of Toxoplasma gondii in blood samples of female sheep and goats in Tebessa, northeastern Algeria.

Toxoplasmosis, caused by Toxoplasma gondii, is a parasitic zoonosis of crucial medical and veterinary importance. It is mainly diagnosed by serological methods which are limited by insufficient sensitivity. Therefore, it is necessary to rely on direct detection of the parasite.

Point mutations within the LdNT2 nucleoside transporter gene from Leishmania donovani confer drug resistance and transport deficiency.

Leishmania donovani, a protozoan parasite, expresses an unusual inosine/guanosine-specific transporter, LdNT2, the gene for which was cloned by functional rescue of a drug-resistant, LdNT2-deficient (FBD5) strain. In this investigation, we have uncovered and characterized the mutations within the LdNT2 open reading frame that are the basis for the drug-resistance and transport-incompetent phenotype of the FBD5 line. The FBD5 cells were shown to be compound heterozygotes in which both mutant ldnt2 alleles harbor discrete point mutations, each of which impaired transport function and conferred resistance to formycin B, the drug to which the clonal FBD5 line was selected.