Publications by authors named "S Buerki"
Article Synopsis
- Delimiting species boundaries is a complex issue in systematics, especially when dealing with subtle morphological variations that can arise from environmental influences or speciation.
- The Lomatium foeniculaceum species complex, widespread in the western U.S. and Canada, has traditionally been classified into five subspecies, but genetic analysis suggests these may be distinct clades.
- Using molecular data from the Angiosperm353 baits kit, the study identified six monophyletic clades and highlights the need for a reevaluation of taxonomic ranks based on both genetic and morphological traits.
Premise: Polyploidization is often followed by diploidization. Diploidization is generally studied using synthetic polyploid lines and/or crop plants, but rarely using extant diploids or nonmodel plants such as Artemisia tridentata. This threatened western North American keystone species has a large genome compared to congeneric Artemisia species; dominated by diploid and tetraploid cytotypes, with multiple origins of tetraploids with genome size reduction.
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- Angiosperms are vital for ecosystems and human life, making it important to understand their evolutionary history to grasp their ecological dominance.
- The study builds an extensive tree of life for about 8,000 angiosperm genera using 353 nuclear genes, significantly increasing the sampling size and refining earlier classifications.
- The findings reveal a complex evolutionary history marked by high gene tree conflict and rapid diversification, particularly during the early angiosperm evolution, with shifts in diversification rates linked to global temperature changes.
Purpose: Genetic studies in sudden infant death syndrome (SIDS) and sudden unexplained death (SUD) cohorts have indicated that cardiovascular diseases might have contributed to sudden unexpected death in 20-35 % of autopsy-negative cases. Sudden unexpected death can also occur in people with epilepsy, termed as sudden unexpected death in epilepsy (SUDEP). The pathophysiological mechanisms of SUDEP are not well understood, but are likely multifactorial, including seizure-induced hypoventilation and arrhythmias as well as genetic risk factors.
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