Publications by authors named "Reun-Ping Goh"

Article Synopsis
  • - In December 2022, a leaf blight disease was observed in 2-month-old pocketbook plants (F1 'Dainty') at a nursery in Ren'ai Township, with about 20% showing symptoms like brown or gray lesions and wilting.
  • - Samples were taken from affected plants, and bacteria isolated from the lesions were identified as Gram-negative, producing fluorescent pigments and able to induce a hypersensitive response in tobacco.
  • - DNA sequencing revealed high similarity to known strains from the GenBank database, confirming the identity of the bacteria and suggesting a connection to specific plant pathogenic strains.
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spp. are phytopathogenic bacteria whose phylogeny has been continuously revised throughout the years. Previous studies on 's phenotypic diversity often analyzed strains obtained from specific crops or adopted outdated classification systems.

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(family Euphorbiaceae) is a leaf ornamental commonly known as variegated croton, which is often found in gardens or grown as indoor plants. In December of 2022, two cutting-propagated variegated croton plants exhibiting abnormal shoot proliferation and little leaf symptoms (Fig. S1) were found in a nursery owned by a private breeder in Wanluan Township, Pingtung County, Taiwan.

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Zucchini yellow mosaic virus (ZYMV) seriously damages cucurbits worldwide. Control of ZYMV by cross-protection has been practised for decades, but selecting useful mild viruses is time-consuming and laborious. Most attenuated potyviruses used for cross-protection do not induce hypersensitive reaction (HR) in Chenopodium quinoa, a local lesion host Chenopodium quinoa.

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Control of plant viruses by cross-protection is limited by the availability of effective protective strains. Incorporation of an NIa-protease processing site in the extreme N-terminal region of the helper component protease (HC-Pro) of turnip mosaic virus (TuMV) resulted in a mutant virus TuHN I that induced highly attenuated symptoms. Recombination analysis verified that two variations, F7I mutation and amino acid 7-upstream-deletion, in HC-Pro co-determined TuHN I attenuation.

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