Transient cerebral ischaemic attacks and calcium-magnesium imbalance: clinical and paraclinical findings in 106 patients under 50 years of age.

One hundred and six selected patients (71 women, 35 men) suffering from transient cerebral ischaemic attacks (TIA) when aged less than 50 years received a comprehensive diagnostic assessment. Patients were classified into three subgroups, into which 9% could be assigned in approximately equal proportions. These were group A; TIA patients with tetanic syndrome (median age of first TIA 32 years, mainly female, 3.

Dental and periodontal disturbances due to magnesium deficit.

Experimental results in female rats with marginal magnesium deficiency a and clinical results in over 100 female tetanic patients suggest the deleterious influence of magnesium on oro-facial tissues.

Effects of trifluoperazine and membrane-bound sialic acid on 45Ca2+ uptake into erythrocytes.

After removal of more than 70% of the sialic acid from the erythrocyte membrane by treatment with neuraminidase, trifluoperazine (TFP) increased the rapid and slow phase of 45Ca2+ uptake into erythrocytes, identically to Ca2+ uptake into untreated erythrocytes from patients with tetanic syndrome. Both phases of 45Ca2+ influx were inhibited by extracellular Mg2+, Ki amounted to 4 mmol/l. However, the increased 45Ca2+ influx into patient erythrocytes in the presence of TFP was not caused by a reduced sialic acid content in the patients' erythrocytes.