Highly oxygenated cyclohexenes from the leaves of Roxb. ex Hornem and their cytotoxic and -glucosidase inhibitory activities.

Phytochemical investigation of the leaf extract of Roxb. ex Hornem led to the isolation and identification of two new highly oxygenated cyclohexenes, uvariagrandols A () and B (), together with seven known compounds (-). Their structures were elucidated by spectroscopic methods as well as comparisons made from the literature.

Population encoding of observed and actual somatosensations in the human posterior parietal cortex.

Cognition relies on transforming sensory inputs into a generalizable understanding of the world. Mirror neurons have been proposed to underlie this process, mapping visual representations of others' actions and sensations onto neurons that mediate our own, providing a conduit for understanding. However, this theory has limitations.

Nigrosporinol Sulfoxides A and B, Sulfur-Bridged Heterocyclic Sesquiterpenoids Produced by Cultures of the Fungus .

The sesquiterpenoids nigrosporinol sulfoxides A () and B () have been isolated from cultures of the endophytic fungus harvested from the sunchoke L. collected in Thailand. Nigrosporinol sulfoxides A () and B () have 4/5/5/5/7 heterocyclic skeletons featuring a sulfoxide bridge not previously found in a terpenoid natural product from any living source.

Rapid response of moss-associated nitrogen fixation to nutrient additions in tropical montane cloud forests with different successional stages.

Unlabelled: Nitrogen (N) fixation in association with mosses could be a key source of new N in tropical montane cloud forests since these forests maintain high humidity levels and stable temperatures, both of which are important to N fixation. Here, nutrient availability could be a prominent control of N fixation processes. However, the mechanisms and extent of these controls, particularly in forests at different successional stages, remains unknown to date.

Muscle Contractility in Hypokalemic Periodic Paralysis.

Introduction/aims: Primary hypokalemic periodic paralysis (HypoPP) can present with periodic paralysis and/or permanent muscle weakness. Permanent weakness is accompanied by fat replacement of the muscle. It is unknown whether the permanent muscle weakness is solely due to fat replacement or if other factors affect the ability of the remaining muscle fibers to contract.