[Experimental steroid osteoporosis in rats and the way to restore the bone tissue structure].

Experimental prednisolone administration has been shown to cause osteoporosis in animals. A single administration of fetal tissue--10-day fetal homogeneous tissue at a dose of 0.5 mg into the experimental animal femur improves the bone tissue structure, hastens its remodeling, contributes to elimination of the bone tissue pathologic process in osteoporosis.

[Protective and restorative effect of antioxidants in experimental intoxication with chlorophenols].

Experimental studies proved that ortho-chlorophenol intoxication is manifested by increased serum markers of cytolysis (alanine aminotransferase, aspartate aminotransferase) and cholestasis (alkaline phosphatase), intensified lipid peroxidation, depressed activity of antioxidant enzymes (catalase), impaired liver functional state. Studies covered protective efficiency of succinate oxymethyluracil and referent medication mexidol for ortho-chlorophenol poisoning.

[The influence of the actoprotectors on lipid peroxidation and erythrocyte membranes in rats poisoned with malathion ].

Actoprotecting properties ofbemitil, tietasol in combination with atropin were studied in red cell membranes and lipid peroxidation of rats poisoned with MI in a dose 320 mg/kg (0.9 LD50). Atropin treatment showed a low effect.