[Fishway design based on the swimming ability of two species in the Yalung River, China].

To investigate the swimming ability of two species in the Yalung River and provide basic parameters for the studies on fish behavior and the design of fish passage, we exa-mined the induced velocity, critical swimming speed, and burst swimming speed in and with incremental velocity method and the durable swimming speed in with fixed velocity method. The results showed that the induced velocity of both species increased first and then plateaued with the increases of body length, with the maximum values being lower than 0.2 m·s.

Factors for self-management activities among rural patients with chronic hepatitis B: A cross-sectional study.

Aims And Objectives: To assess the self-management activities among rural patients with chronic hepatitis B (CHB), and the influence of psychosocial and demographic factors on their self-management activities.

Background: Chronic hepatitis B is a serious public health concern. Rural patients may have limited access to healthcare services.

Self-management behaviors in adults with chronic hepatitis B: A structural equation model.

Background: Chronic hepatitis B is a serious and chronic health problem, requiring self-management to control the disease and related complications.

Objectives: To develop a structural model to identify how social support, self-efficacy and disease knowledge contribute to their self-management behaviors in adults with chronic hepatitis B.

Design: A cross-sectional study.

Gene expression profile after knockdown of USP18 in Hepg2.2.15 cells.

In our previous work, we found that the expression of ubiquitin-specific protease 18 (USP18), also known as UBP43, is associated with the efficiency of interferon alpha (IFN-α) treatment in patients with chronic hepatitis B (CHB). To elucidate the influence of USP18 on hepatitis B virus (HBV) replication and the mechanism of this activity, we silenced USP18 by introducing short hairpin RNA (shRNA) into Hepg2.2.

Correction: Suppression of USP18 Potentiates the Anti-HBV Activity of Interferon Alpha in HepG2.2.15 Cells via JAK/STAT Signaling.

[This corrects the article DOI: 10.1371/journal.pone.

Suppression of USP18 Potentiates the Anti-HBV Activity of Interferon Alpha in HepG2.2.15 Cells via JAK/STAT Signaling.

Ubiquitin-specific protease 18 (USP18, also known as UBP43) has both interferon stimulated gene 15 (ISG15) dependent and ISG15-independent functions. By silencing the expression of USP18 in HepG2.2.