Endocarditis Mimicking Crescentic Glomerulonephritis with PR3-ANCA Positivity.

is a fastidious organism that causes cat scratch disease, commonly associated with fever and lymphadenopathy but, in rare instances, also results in culture-negative infectious endocarditis. We describe a patient who presented with flank pain, splenic infarct, and acute kidney injury with an active urinary sediment, initially suspicious for vasculitis, which was subsequently diagnosed as endocarditis. endocarditis may present with a crescentic glomerulonephritis (GN) and elevated PR3-ANCA antibody titers, mimicking ANCA-associated GN, with 54 cases reported in the literature.

Bilateral Testicular Infarction from IgA Vasculitis of the Spermatic Cords.

A 51-year-old man with type 2 diabetes mellitus and chronic obstructive pulmonary disease presented to the emergency room with increasing bilateral leg pain, rash, and scrotal swelling with pain. Skin biopsy from his thigh revealed IgA-associated vasculitis. Due to hematuria, a renal biopsy was performed and showed an IgA glomerulonephritis with focal fibrinoid necrosis and neutrophil accumulation.

Stent placement versus angioplasty improves patency of arteriovenous grafts and blood flow of arteriovenous fistulae.

Background: While endovascular stent placement is the standard of care in most percutaneous coronary and peripheral artery intervention, its role in the salvage of thrombosed and stenotic hemodialysis access remains controversial.

Design, Setting, Participants, And Measurements: We compared the effects of stent versus angioplasty on primary patency rates in the treatment of stenotic arteriovenous fistulae (AVF) and arteriovenous grafts (AVGs). Moreover, we compared access flow (Qa) and urea reduction ratio (URR) between the two groups as a metric of the effect of stent placement versus angioplasty on dialysis delivery.

Pseudohyperkalemia due to pneumatic tube transport in a leukemic patient.

Pseudohyperkalemia is an elevation in measured serum or plasma potassium levels caused by mechanical release of potassium from cells during phlebotomy or specimen processing. We describe a case of pseudohyperkalemia caused by mechanical disruption of white blood cells from a leukemic patient because of pneumatic tube transport of the specimen. This is the first description of pneumatic tube transport causing pseudohyperkalemia, and clinicians should be aware of this potential cause of false elevation of plasma potassium levels.