Publications by authors named "Pablo Ruiz-Hernandez"

Introduction And Objectives: We report the results of the 2023 Spanish catheter ablation registry.

Methods: Procedural data were collected and incorporated into the REDCap platform by all participating centers through a specific form.

Results: There were 104 participating centers in 2023 compared with 103 in 2022.

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Article Synopsis
  • Clinical data indicate that cardiosphere-derived cells (CDCs) could potentially improve heart recovery following a heart attack by altering scar tissue and reducing the risk of ventricular tachycardia (VT).
  • In a study on pigs with induced heart attacks, CDC treatment was associated with improved conduction speed and longer action potential duration, leading to fewer instances of induced VT compared to a control group.
  • Histological analysis of the treatment group showed reduced fibrosis and a higher density of connexin-43, suggesting that CDCs may enhance the electrical and structural properties of the heart after injury, highlighting their potential as a new therapy for heart attack recovery.
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IntroducciÓn Y Objetivos: La tormenta eléctrica (TE) se caracteriza por episodios repetidos de taquicardia ventricular o fibrilación ventricular relacionados con mal pronóstico a corto y largo plazos. El objetivo fue evaluar la prevalencia, resultados y supervivencia de los pacientes sometidos a tratamiento intervencionista por TE en un centro de referencia.

MÉtodos: Estudio unicéntrico, observacional y retrospectivo.

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Introduction And Objectives: The development of complete AV block and the need for pacemaker implantation (PM) is the most frequent complication after Transaortic valve replacement (TAVR). In other PM clinical contexts, a higher percentage of ventricular stimulation has been associated with worse prognosis. The objective was to study the existence of predictors of PM dependence.

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Background: Catecholaminergic polymorphic ventricular tachycardia (CPVT) is a difficult-to-diagnose cause of sudden cardiac death (SCD). We identified a family of 1400 individuals with multiple cases of CPVT, including 36 SCDs during youth.

Objectives: We sought to identify the genetic cause of CPVT in this family, to preventively treat and clinically characterize the mutation-positive individuals, and to functionally characterize the pathogenic mechanisms of the mutation.

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