Distributional justice and climate risk assessment: An analysis of disparities within direct and indirect risk.

Climate change and natural hazard risk assessments often overlook indirect impacts, leading to a limited understanding of the full extent of risk and the disparities in its distribution across populations. This study investigates distributional justice in natural hazard impacts, exploring its critical implications for environmental justice, equity, and resilience in adaptation planning. We employ high-resolution spatial risk assessment and origin-destination routing to analyze coastal flooding and sea-level rise scenarios in Aotearoa New Zealand.

Climate change adaptation through an integrative lens in Aotearoa New Zealand.

Climate change is being felt across all human and natural systems in Aotearoa New Zealand and is projected to worsen this decade as impacts compound and cascade through natural system and sectoral dependencies. The effectiveness of adaptation is constrained by how fast greenhouse gas emissions are reduced globally, the pace of change, the frequency and progression of impacts, and the capacity of our natural, societal and political systems to respond. We explore how these systems and sectors interact with existing and projected climate change stressors by categorising climate change impacts (Trends and Events) and consequential thresholds (Thresholds), and by grouping systems and sectors by types (Typologies).

Ethnic differences in ApoC-III concentration and the risk of cardiovascular disease: No evidence for the cardioprotective role of rare/loss of function variants in non-Europeans.

Background: Hypertriglyceridemia is as an independent risk factor for cardiovascular disease (CVD). Apolipoprotein C-III (ApoC-III) is known to regulate triglyceride (TG) metabolism. However, the causal association between ApoC-III and CVD development is unclear.

APOC3 genetic variation, serum triglycerides, and risk of coronary artery disease in Asian Indians, Europeans, and other ethnic groups.

Background: Hypertriglyceridemia has emerged as a critical coronary artery disease (CAD) risk factor. Rare loss-of-function (LoF) variants in apolipoprotein C-III have been reported to reduce triglycerides (TG) and are cardioprotective in American Indians and Europeans. However, there is a lack of data in other Europeans and non-Europeans.