Comparative effectiveness of acupoint stimulation for preventing postoperative nausea and vomiting after general anesthesia: A network meta-analysis of randomized trials.

Objective: To systematically evaluate the effectiveness of different acupoint stimulation techniques in preventing postoperative nausea and vomiting (PONV) after general anesthesia.

Methods: We searched PubMed, Cochrane Library, Web of Science, Embase for relevant papers, about the effect of acupoint stimulation for preventing PONV from their inception to July 31, 2023. Two reviewers performed study screening, data extraction, and risk of bias assessment.

Regulable crack patterns for the fabrication of high-performance transparent EMI shielding windows.

Crack pattern-based metal grid film is an ideal candidate material for transparent electromagnetic interference shielding optical windows. However, achieving crack patterns with narrow grid spacing, small wire width, and high connectivity remains challenging. Herein, an aqueous acrylic colloidal dispersion was developed as a crack precursor for preparing crack patterns.

Plastome data provides new insights into population differentiation and evolution of Ginkgo in the Sichuan Basin of China.

Background: Ginkgo biloba L., an iconic living fossil, challenges traditional views of evolutionary stasis. While nuclear genomic studies have revealed population structure across China, the evolutionary patterns reflected in maternally inherited plastomes remain unclear, particularly in the Sichuan Basin - a potential glacial refugium that may have played a crucial role in Ginkgo's persistence.

Glycogen synthase GYS1 overactivation contributes to glycogen insolubility and malto-oligoglucan-associated neurodegenerative disease.

Polyglucosans are glycogen molecules with overlong chains, which are hyperphosphorylated in the neurodegenerative Lafora disease (LD). Brain polyglucosan bodies (PBs) cause fatal neurodegenerative diseases including Lafora disease and adult polyglucosan body disease (ABPD), for which treatments, biomarkers, and good understanding of their pathogenesis are currently missing. Mutations in the genes for the phosphatase laforin or the E3 ubiquitin ligase malin can cause LD.