Publications by authors named "P L Acutis"

has been considered for a long time the only causal agent of lactococcosis. In recent years, different papers reported the involvement of other two bacterial species: and . A different host tropism has been described for these species where and are predominant species in fish and humans' infections, while in bovine.

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Herpesvirus (HV) is widely distributed among cetacean populations, with the highest prevalence reported in the Mediterranean Sea. In this study, a comprehensive analysis was conducted, including epidemiological, phylogenetic, and pathological aspects, with particular emphasis on neuropathology, to better understand the impact of HV in these animals. Our results show a higher presence of HV in males compared to females, with males exhibiting a greater number of positive tissues.

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Article Synopsis
  • Oysters are bivalve molluscs crucial for fisheries and aquaculture, but their genetic diversity and distribution are still not well understood.
  • The variability in their shell shapes makes it hard to classify different species, despite molecular research identifying about 100 existing species.
  • This study focused on small flat oysters in the Mediterranean regions of Liguria and Sardinia, using 16S rRNA sequencing to reveal new species and enhance knowledge about oyster diversity and evolution.
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After the detection of bovine spongiform encephalopathy (BSE), and a zoonotic transmissible spongiform encephalopathy (TSE) caused by the pathological prion protein (PrP) in two goats, the investigation of goat prions became of greater interest. Therefore, a broad collection of European goat TSE isolates, including atypical scrapie, CH1641 and goat BSE as reference prion strains were biochemically characterised and subsequently inoculated into seven rodent models for further analysis (already published results of this comprehensive study are reviewed here for comparative reasons). We report here the histopathological and immunohistochemical data of this goat TSE panel, obtained after the first passage in Tgshp IX (tg-shARQ) mice, which overexpress the ovine prion protein.

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