[Functional morphology of the myocardium in acute experimental peritonitis].

Structural disorganization of the myocardium starts at the very early stages of acute experimental peritonitis, the maximum surface of the contractural lesions is registered one day and the maximum decrease of the adrenergic nervous fiber density 6 hrs after the start of the experiment. The change of the mitochondria surface and the biochemical process intensity in the cardiomyocytes depend on the disease stage. In the process of recovery the cardiomyocyte and microcirculatory bed repair starts to dominate.

[Iatrogenic pathology, its role in the structure of patient morbidity and thanatogenesis].

Iatrogenic diseases which were an immediate cause of death or played an important role in thanatogenesis were found in 319 (14.2%) of 2250 autopsy cases of patients with surgical, therapeutical and neurosurgical pathology. Surgical iatrogenesis was established in 226 (70.