Affiliation in times of pandemics: Determinants and consequences.

Affiliation is a basic human need, especially during difficult times. To what extent did the need to affiliate limit our capacity to abide by health guidelines, in particular regarding social distancing, during the COVID-19 pandemic? We investigated this issue using questionnaire data from two samples of the French population collected during the first French lockdown (April-May 2020). We found that in men, higher social comparison orientation (sensitivity to the needs of others and inclination to help) and higher perceived threat increased the frequency of reported affiliative activities.

Experience and memory of time and emotions two years after the start of the COVID-19 pandemic.

In this French longitudinal study, we assessed judgment of the passage of time in current life and the predictors of this judgment 2 years after the onset of the COVID-19 pandemic, i.e., at a time when there was no lockdown and no protective measures.

Joint action with human and robotic co-actors: Self-other integration is immune to the perceived humanness of the interacting partner.

When performing a joint action task, we automatically represent the action and/or task constraints of the co-actor with whom we are interacting. Current models suggest that, not only physical similarity, but also abstract, conceptual features shared between self and the interacting partner play a key role in the emergence of joint action effects. Across two experiments, we investigated the influence of the perceived humanness of a robotic agent on the extent to which we integrate the action of that agent into our own action/task representation, as indexed by the Joint Simon Effect (JSE).

Microglial phagocytosis dysfunction in stroke is driven by energy depletion and induction of autophagy.

Microglial phagocytosis of apoptotic debris prevents buildup damage of neighbor neurons and inflammatory responses. Whereas microglia are very competent phagocytes under physiological conditions, we report their dysfunction in mouse and preclinical monkey models of stroke (macaques and marmosets) by transient occlusion of the medial cerebral artery (tMCAo). By analyzing recently published bulk and single cell RNA sequencing databases, we show that the phagocytosis dysfunction was not explained by transcriptional changes.