Monitoring exposure of brown bullheads and benthic macroinvertebrates to sediment contaminants in the Ashtabula River before, during, and after remediation.

In 2007, approximately 420,500 cubic meters of contaminated sediment were removed from the Ashtabula River by dredging. The primary objective of the present study was to monitor contaminant exposure in fish and macroinvertebrates before, during, and after dredging. This was done by measuring tissue concentrations of polychlorinated biphenyl (PCB) and polycyclic aromatic hydrocarbon (PAH) in brown bullhead catfish (Ameriurus nebulosa) and in benthic macroinvertebrates, assessing changes in DNA damage in fish liver and blood, and scoring external and histopathological lesions and anomalies in the fish.

An integrated assessment of sediment remediation in a midwestern U.S. stream using sediment chemistry, water quality, bioassessment, and fish biomarkers.

A comprehensive biological, sediment, and water quality study of the lower Little Scioto River near Marion, Ohio, USA, was undertaken to evaluate the changes or improvements in biotic measurements following the removal of creosote-contaminated sediment. The study area covered 7.5 river miles (RMs), including a remediated section between RMs 6.

Feasibility of micronucleus methods for monitoring genetic damage in two feral species of small mammals.

Peromyscus leucopus (white-footed mouse) and Cryptotis parva (least shrew) possess desirable attributes for biomonitoring contamination of terrestrial ecosystems, but few studies have examined the potential use of these species for monitoring exposure to genotoxic contaminants. The susceptibility of laboratory-reared C. parva, P.

New nephron development in fish from polluted waters: a possible biomarker.

: Recent evidence has shown that fish have the ability to develop new nephrons following renal injury. This study evaluated the usefulness of quantifying developing nephrons in mature fish as an ecotoxicological assessment tool. Histological sections of kidney were prepared from Atlantic tomcod and brown bullhead specimens collected from reference and contaminated streams.

Species and strain sensitivity to the induction of peroxisome proliferation by chloroacetic acids.

B6C3F1 mice and Sprague-Dawley rats were provided drinking water containing 6-31 mM (1-5 g/liter) trichloroacetic acid (TCA), 8-39 mM (1-5 g/liter) dichloroacetic acid (DCA), or 11-32 mM (1-3 g/liter) monochloroacetic acid (MCA) for 14 days. TCA and DCA, but not MCA, increased the mouse relative liver weight in a dose-dependent manner. Rat liver weights were not altered by TCA or DCA treatment, but were depressed by MCA.