Birth Defects Res B Dev Reprod Toxicol
April 2008
Background: In this study, the mutated phenotypes were produced by treatment of chemical mutagen, N-ethyl-N-nitrosourea (ENU). We analyzed the mutated mice showing the specific phenotype of ectodermal dysplasia (ED) and examined the affected gene.
Methods: Phenotypes, including size, bone formation, and craniofacial morphology of ENU-induced ED mice, were focused.
Thromb Haemost
February 1994
Rheumatoid arthritis is a chronic inflammatory disease caused essentially by an immune-mediated mechanism. However, abnormalities of the clotting system have also been incriminated as having an important role in the pathogenesis of this disease. This study aims at assessing the clotting system and collagen metabolism alterations and the relationship between perturbances of the hemostatic pathway and the destructive and fibroproliferative processes in patients with rheumatoid arthritis.
View Article and Find Full Text PDFA method of thin layer chromatographic separation and ultraviolet spectrophotometric determination of tangshenoside I in Codonopsis pilosula is described. A comparison of the contents in various samples is made. The contents of tangshenoside I in frosted sample have been found to be twice as much as in normal drug.
View Article and Find Full Text PDFEighty-two ultrasonographic examinations were performed on 60 of our pregnant patients with regular menstrual cycles and no complications. The pregnancies ranged from 7 to 13 weeks of gestation. Thoracic anteroposterior diameter (ETAPD), transverse diameter (ETTD), cross-sectional area (ETA) and crown-rump length (CRL) were measured on each ultrasonogram.
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