[Splenic infarction and abscess as a complication of atrial fibrillation].

The case of a 64 year old man is presented by the authors whose spleen infarction developed in connection with atrial fibrillation was transformed into splenic abscess in spite of the applied conservative treatment. According to the overviewed literature the etiology, symptoms, diagnostic and therapeutic possibilities, as well as prognosis of these two entities related to spleen are described hereby.

Protection of functional and structural integrity of the rat myocardium by dexamethasone in hemorrhagic shock.

Rat hearts were isolated from control animals (C), following 2.5 hours of hemorrhagic hypotension (Sh) and from rats pretreated with 10 mg/kg dexamethasone (Sh + D), and they were perfused by the modified Langendorff technique. Dexamethasone pretreatment prevented the development of depression of left ventricular mechanical performance and increase of coronary resistance, but the reduction of O2 consumption observed in Sh hearts was not influenced by the synthetic glucocorticoid.

[Effect of dexamethasone on the ultrastructure of the myocardium in experimental hemorrhagic shock].

Authors studied the ultrastructure of the myocardium of rats treated with Dexamethasone in experimental haemorrhagic shock of 2.5 hours duration. The perfusion was performed on the Langendorff cannula.

Shock-induced cytoplasmic NADH fluorescence changes in the living cat brain cortex: effect of dexamethasone.

The effect of dexamethasone administration on cerebrocortical NADH fluorescence, blood flow and blood volume, intracellular oxygen tension and electrocorticogram (ECoG) was studied in anaesthetized cats in two sets of experiments. Haemorrhagic shock was induced by stepwise decrease of mean arterial blood pressure from the control level a to 80, 60 and 40 mmHg. The bleeding was followed by reinfusion of the shed blood.