[Morphological changes caused by the dermonecrosis factor of the causative agents of cholera and various other intestinal infections].

Intra- or subepithelial focal purulent inflammation with necrosis of exudating leucocytes during 1-2 days is developed in consequence of intradermal injection of the living cholera vibrios, cultured on membrane agar, or their supernatants. Sometimes coagulative necrosis of cover epithelium arises without preliminary purulent inflammation stage from the very beginning. Intradermal injection of living cholera vibrios leads to the development of coagulative necrotic foci in derma too.

[Serological varieties of Klebsiella pneumoniae in pneumonia].

118 K. pneumoniae cultures isolated from the pulmonary tissue samples, taken during autopsy from 33 patients who had died of pneumonia, and from the sputum of 19 living patients with chronic pneumonia, as well as 2 strains, K1 and K2, from the International Reference Klebsiella collection, formerly known as Friedländer's bacilli A and B, were studied. Besides K.

[Enzymomorphology in cholerogenic intoxication in suckling rabbits].

The cholerogenic effect produced in suckling rabbits by intraintestinal administration of a cholerogen was shown to be accompanied by the edema of villi, formation of lympho-histiocytic infiltrates in the stroma and bases of crypts, hemorrhages in the stroma of the villi and loss of individual enterocytes mostly on tops of the villi. In the large intestine, vacuolation and pyknosis of the epithelial nuclei and edema of the submucosa were observed. The intensity of the cholerogenic effect correlated with the degree of the decline of the activity of succinate dehydrogenase, adenosine triphosphatase, alkaline and acid phosphatases in the intestinal tract.