type III effector AopU interferes with plant immune responses and interacts with a watermelon E3 ubiquitin ligase.

is a seed-borne bacterium that causes bacterial fruit blotch of watermelon and other cucurbit plants worldwide. It uses a type III secretion system to inject type III effectors (T3Es) into plant cells, which affect the host immune responses and facilitate pathogen colonization. However, the current understanding of the specific molecular mechanisms and targets of these effectors in is limited.

Complete Genome Sequence of Erwinia amylovora Strain 99east-3-1, Isolated from Pyrus sinkiangensis in China.

Here, we report the complete genome sequence of Erwinia amylovora strain 99east-3-1, which was isolated from Pyrus sinkiangensis in Xinjiang Uygur Autonomous Region, China.

Effector AopV Suppresses Plant Immunity and Interacts with Aromatic Dehydratase ADT6 in Watermelon.

Bacterial fruit blotch (BFB) is a disease of cucurbit plants caused by . Although has great destructive potential, the molecular mechanisms of pathogenicity of are not clear, particularly with regard to its type III secreted effectors. In this study, we characterized the type III secreted effector protein, AopV, from strain Aac5.

Hcp of the Type VI Secretion System (T6SS) in Group II Strain Aac5 Has a Dual Role as a Core Structural Protein and an Effector Protein in Colonization, Growth Ability, Competition, Biofilm Formation, and Ferric Iron Absorption.

A type VI secretion system (T6SS) gene cluster has been reported in . Research on the activation conditions, functions, and the interactions between key elements in . T6SS is lacking.

Essential Virulence Gene Activates Host Immune Response against Pathogen.

Bacterial fruit blotch (BFB) caused by (Ac) is a devastating watermelon disease that severely impacts the global watermelon industry. Like other Gram-negative bacteria, the type three secretion system (T3SS) is the main pathogenicity factor of . The T3SS apparatus gene codes for the Hrp pilus and serves as a conduit to secret effector proteins into host cells.