Explanatory factors of digital citizenship among university students. A cross-national dataset.

The present research explores a set of explanatory factors of digital citizenship in university students on a cross-national sample. The data were collected from 501 university students aged 19-23 years old, from two public universities in Romania ( = 350) and Italy ( = 151). Information was gathered from one university in each country by using an online survey form.

Evidence to the Need for a Unifying Framework: Critical Consciousness and Moral Education in Adolescents Facilitate Altruistic Behaviour in the Community.

Background: Critical consciousness represents an emancipatory pedagogical process whose central goal is developing the necessary skills to identify and act in the direction of changing social limitations. An important kind of action that helps challenge social limitations is altruistic behaviour. Moreover, moral values could enhance the effect of critical consciousness on altruistic behaviour.

How personal values and critical dispositions support digital citizenship development in higher education students.

The virtual environment's expansion and role in young people's lives accentuate the need for developing transversal competences such as digital citizenship. The process may be supported by personal resources like personal values and critical thinking dispositions. With this study on 536 young students' students aged 18 to 26 ( = 20.

Microarray Analysis of Gene Expression Reveals that Cyclo-oxygenase-2 Gene Therapy Up-regulates Hematopoiesis and Down-regulates Inflammation During Endochondral Bone Fracture Healing.

Background: Cyclo-oxygenase-2 (Cox-2) is an inflammatory mediator that is necessary for the tissue repair, including bone fracture healing. Although the application of Cox-2 gene therapy to a murine closed femoral fracture has accelerated bony union, but the beneficial effect was not observed until the endochondral stage of bone repair that is well after the inflammatory stage normally subsides.

Methods: To identify the molecular pathways through which Cox-2 regulates fracture healing, we examined gene expression profile in fracture tissues in response to Cox-2 gene therapy during the endochondral bone repair phase.

Urokinase plasminogen activator gene deficiency inhibits fracture cartilage remodeling.

Urokinase plasminogen activator (uPA) regulates a proteolytic cascade of extracellular matrix degradation that functions in tissue development and tissue repair. The development and remodeling of the skeletal extracellular matrix during wound healing suggests that uPA might regulate bone development and repair. To determine whether uPA functions regulate bone development and repair, we examined the basal skeletal phenotype and endochondral bone fracture repair in uPA-deficient mice.