Cutaneous inflammation is recurrent in systemic lupus erythematosus (SLE), yet mechanisms that drive cutaneous inflammation in SLE are not well defined. Type I IFNs are elevated in nonlesional SLE skin and promote inflammatory responses. Staphylococcus aureus, known to induce IFN production, could play a role in cutaneous inflammation in SLE.
View Article and Find Full Text PDFChromagar is a medium that is used in culture-based colonization studies of Staphylococcus aureus. We have found that S. aureus negative colonies often fit the color recommendations for S.
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