Publications by authors named "Nathan Egnew"
Coping strategies in response to different levels of elevated water hardness in channel catfish (Ictalurus punctatus): Insight into ion-regulatory and histopathological modulations.
Comp Biochem Physiol A Mol Integr Physiol
October 2021
Water hardness above the optimal level can incite toxic effects in fish, which are often species specific. Hence, we aimed at obtaining insights on the potential effects of elevated water hardness as well as coping strategies in channel catfish (Ictalurus punctatus). First, a toxicity assay was performed where the 96 h-LC was calculated as 4939 mg/L CaCO.
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- Iron overload in water is a major environmental issue and negatively affects largemouth bass, an important sport and aquaculture fish.
- A 96-hour toxicity test established a tolerance limit of 22.07 mg/L iron for the fish, and longer-term exposure to a lower concentration (5.52 mg/L) led to significant physiological changes and impaired metabolic functions over 28 days.
- Findings indicate that chronic exposure to high iron levels disrupts ion balance, affects ammonia excretion, and depletes energy reserves in the fish, highlighting the risks of waterborne iron overload to aquatic ecosystems.
Iron overload in water is a problem in many areas of the world, which could exert toxic effects on fish. To achieve maximum growth and overall fitness, iron induced toxicity must be alleviated. Therefore, this research was undertaken to investigate the potential mitigation of iron toxicity by dietary vitamin C supplementation in channel catfish (Ictalurus punctatus).
View Article and Find Full Text PDFGoldfish (Carassius auratus) juveniles were exposed to virgin polyvinyl chloride microplastics (PVC-MPs) in triplicate at 0, 0.1 or 0.5 mg/L for four days.
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- Waterborne ammonia is harmful to aquatic animals, but the effects on largemouth bass, a valuable species in U.S. fisheries, are not thoroughly understood.
- A 96-hour toxicity test revealed the lethal concentration of ammonia for largemouth bass and subsequent research assessed physiological and molecular responses to prolonged exposure to high environmental ammonia (HEA).
- Findings showed that after 28 days of HEA exposure, the fish could efficiently regulate ammonia excretion, indicating a compensatory mechanism, and there were notable changes in ion regulation and gene expression linked to ammonia tolerance.