Rat liver mitochondrial damage under acute or chronic carbon tetrachloride-induced intoxication: protection by melatonin and cranberry flavonoids.

In current societies, the risk of toxic liver damage has markedly increased. The aim of the present work was to carry out further research into the mechanism(s) of liver mitochondrial damage induced by acute (0.8 g/kg body weight, single injection) or chronic (1.

[Hepatoprotective effects of amino acids with branched hydrocarbon chains and taurine in experimental subchronic alcohol intoxication and ethanol withdrawal].

Forced alcoholization of rats according to Majchrowicz led to the development of fatty liver. The subsequent ethanol withdrawal was accompanied by amino acid imbalance in the pool of free sulfur-containing and glycogenic amino acids in liver and blood plasma. Similar changes were observed after prolonged alcohol intoxication.

[Visceral pathology in acute destructive pancreatitis].

It is suggested that combined therapy of acute destructive pancreatitis should allow for multiprofile visceral pathology induced by pancreatogenic endotoxicosis and intoxication due to secondary purulent infection.

[Are-related morphometric characteristics of the human pancreas].

Quantitative characteristics of the parenchyma and stroma of the pancreas has been determined at the age of 3 days up to 92 years. The data obtained concretize the character and dynamics of involutive changes. To take them into consideration is necessary at estimation of various pathological processes, revealed in the pancreas.