Background: Hydrogen sulfide (HS) has been identified as the third gaseous signaling molecule. Endogenous HS plays a key role in the progression of various types of cancer. However, the effect of endogenous HS on the growth of esophageal cancer (EC) remains unknown.
View Article and Find Full Text PDFHydrogen sulfide (H S) has been widely recognized as one of gasotransmitters. Endogenous H S plays a crucial role in the progression of cancer. However, the effect of endogenous H S on the development of nasopharyngeal carcinoma (NPC) is still unknown.
View Article and Find Full Text PDFThe role of hydrogen sulphide (H S) in angiogenesis has been widely demonstrated. Vascular endothelial growth factor (VEGF) plays an important role in H S-induced angiogenesis. H S promotes angiogenesis by upregulating VEGF via pro-angiogenic signal transduction.
View Article and Find Full Text PDFNasopharyngeal carcinoma (NPC) is an epithelia-derived malignancy with a distinctive geographic distribution. Cystathionine γ-lyase (CSE) is involved in cancer development and progression. Nevertheless, the role of CSE in the growth of NPC is unknown.
View Article and Find Full Text PDFThyroid cancer is considered to be one of the most common endocrine tumors worldwide. Cystathionine -synthase (CBS) plays a crucial role in the occurrence of several types of malignancies. And yet, the mechanism of action of CBS in the growth of thyroid carcinoma cells is still unrevealed.
View Article and Find Full Text PDFHydrogen sulfide (HS) has long been considered as a toxic gas, but as research progressed, the idea has been updated and it has now been shown to have potent protective effects at reasonable concentrations. HS is an endogenous gas signaling molecule in mammals and is produced by specific enzymes in different cell types. An increasing number of studies indicate that HS plays an important role in cardiovascular homeostasis, and in most cases, HS has been reported to be downregulated in cardiovascular diseases (CVDs).
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