Host-microbiota interactions and responses of infected with decapod iridescent virus 1.

Introduction: Decapod iridescent virus 1 (DIV1) has caused severe economic losses in shrimp aquaculture. So far, Researchs on DIV1-infected shrimp have mainly focused on the hemocytes immune response, while studies on the host-intestine microbiota interactions during DIV1 infection have been scarce.

Methods: This study determined the lethal concentration 50 (LC) of DIV1 to , preliminarily determining that could serve as a susceptible object for DIV1.

Integrated analysis of intestinal microbiota and metabolomic reveals that decapod iridescent virus 1 (DIV1) infection induces secondary bacterial infection and metabolic reprogramming in .

In recent years, with global warming and increasing marine pollution, some novel marine viruses have become widespread in the aquaculture industry, causing huge losses to the aquaculture industry. Decapod iridescent virus 1 (DIV1) is one of the newly discovered marine viruses that has been reported to be detected in a variety of farmed crustacean and wild populations. Several previous studies have found that DIV1 can induce Warburg effect-related gene expression.

The Molecular Mechanism of Hemocyte Immune Response in Infected With Decapod Iridescent Virus 1.

As a new type of shrimp lethal virus, decapod iridescent virus 1 (DIV1) has caused huge economic losses to shrimp farmers in China. Up to now, DIV1 has been detected in a variety of shrimps, but there is no report in . In the current study, we calculated the LC to evaluate the toxicity of DIV1 to and determined through nested PCR that can be the host of DIV1.