Publications by authors named "Mark Colin"
Three Hordeum spontaneum-derived resistances (referred to as 145L2, 41T1 and 40Y5) have demonstrated long-term effectiveness against barley scald, caused by Rhynchosporium commune, in western Canada. Genetic mapping of these resistances in three populations, and the use of five barley genome assemblies, revealed they co-located to a narrowly defined 0.58-1.
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- Barley loose smut is largely controlled by a specific gene, but certain inoculation methods can lead to poor seed quality and germination issues, especially in carrier and susceptible genotypes.
- Research compared a carrier strain (TR11698) and a susceptible strain (CDC Austenson), finding that lower inoculum concentrations improved seed traits and increased infection in the susceptible strain, while the carrier showed no infection but still had low germination rates.
- The study suggests that lowering inoculum levels can enhance seed appearance and germination in susceptible plants, while the carrier's seedling issues indicate a resistance mechanism, with increased abscisic acid levels potentially aiding in pathogen survival.
Article Synopsis
- The Un8 gene in barley provides resistance against the Ustilago nuda pathogen, responsible for true loose smut, and is located on the long arm of chromosome 5.
- Recent genetic studies have narrowed the location of the Un8 gene to a small interval on chromosome 1HL, using a population of 4625 segregating lines.
- Analysis of the candidate gene from resistant versus susceptible barley lines revealed specific amino acid variations in the deduced protein that might contribute to the loss of function in susceptible lines.