Publications by authors named "Mari Ebine"

Article Synopsis
  • The superior vena cava (SVC) is linked to initiating atrial fibrillation (AF) by contributing to electrical disturbances that don't involve pulmonary veins.
  • In a study with 47 patients, researchers used electroanatomical mapping to measure the sleeve lengths of pulmonary veins (PVs) and the SVC, discovering that SVC and left superior PV sleeves were longer than others.
  • Findings suggest that left-sided PVs have a connection to patients' body surface area, while there’s a correlation between the sleeve lengths of right-sided PVs and the SVC, offering new insights into AF mechanisms.
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Purpose: Despite the use of steroid-eluting leads, a transient but not persistent rise in the atrial/ventricular capture threshold (TRACT/TRVCT) can occur early after pacemaker implantation in patients with sick sinus syndrome. This study aimed to assess the prevalence, predictors, and mechanisms of TRACT/TRVCT in patients with heart failure undergoing implantable cardioverter defibrillator (ICD) or cardiac resynchronization therapy (CRT) implantation.

Method: One hundred twenty consecutive patients underwent ICD (N = 70) or CRT (N = 50) implantation.

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Background: Steroid-eluting pacemaker leads suppress acute rises in pacing threshold by preventing inflammatory processes. However, we occasionally encounter not persistent but transient rise in the atrial capture threshold (TRACT) early after pacemaker implantation. We believe that this phenomenon is underrecognized in clinical practice and may potentially lead to unnecessary reintervention.

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Introduction: The superior vena cava (SVC) is a main source of nonpulmonary vein (PV) ectopies initiating atrial fibrillation (AF). Empiric SVC isolation may improve rhythm outcomes after catheter ablation of AF. Because the SVC passes immediately adjacent to the right superior PV (RSPV), an electrophysiological relation could be present between the two structures.

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Left ventricular diastolic dysfunction in hypertrophic cardiomyopathy (HC) increases susceptibility to atrial fibrillation. Although phenotypical characteristics of the hypertrophied left ventricle are clear, left atrial (LA) and pulmonary venous (PV) remodeling has rarely been investigated. This study aimed to identify differences in LA and PV remodeling between HC and hypertensive heart disease (HHD) using 3-dimensional computed tomography.

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