Quantifying the resolution of spatial and temporal representation in children with 22q11.2 deletion syndrome.

Objectives: Our ability to generate mental representation of magnitude from sensory information affects how we perceive and experience the world. Reduced resolution of the mental representations formed from sensory inputs may generate impairment in the proximal and distal information processes that utilize these representations. Impairment of spatial and temporal information processing likely underpins the non-verbal cognitive impairments observed in 22q11.

Disrupted fornix integrity in children with chromosome 22q11.2 deletion syndrome.

The fornix is the primary subcortical output fiber system of the hippocampal formation. In children with 22q11.2 deletion syndrome (22q11.

Children with chromosome 22q11.2 deletion syndrome exhibit impaired spatial working memory.

Individuals with chromosome 22q11.2 deletion syndrome (22q11.2DS) have been shown to have impairments in processing spatiotemporal information.

Impaired multiple object tracking in children with chromosome 22q11.2 deletion syndrome.

Background: Chromosome 22q11.2 Deletion Syndrome (22q11.2DS) occurs in approximately 1:4,000 live births with a complex and variable presentation that includes medical, socioemotional and psychological symptoms with intellectual impairment.

A cross-sectional study of the development of volitional control of spatial attention in children with chromosome 22q11.2 deletion syndrome.

Background: Chromosome 22q11.2 deletion syndrome (22q11.2DS) results from a 1.