J Gastroenterol Hepatol
July 2007
Background: Presence or absence of nodal metastasis influences outcome of gastric cancer patients. This study gives insight into survival predictors and clinicopathological features of node-negative gastric adenocarcinoma.
Methods: Between 1988 and 1999, 689 gastric cancer patients without other cancer or gastrectomy for benign disease who underwent curative resection were enrolled in this study.
Hepatogastroenterology
August 2007
Background/aims: Borrmann type I gastric cancers are rare. Its clinicopathological features have never been reported.
Methodology: A total of 33 patients with Borrmann type I gastric cancer was evaluated.
Background/aims: Some gastric cancer patients have multiple primary cancers (MPC). We evaluate the current status of MPC with gastric cancer.
Methodology: 2,109 gastric cancer patients treated between 1987 and 2002 were analyzed.
Background: The survival benefit and morbidity after nodal dissection for gastric cancer remains controversial. We aimed to do a single-institution randomised trial to compare D1 (ie, level 1) lymphadenectomy with that of D3 (ie, levels 1, 2, and 3) dissection for gastric cancer in terms of overall survival and disease-free survival.
Methods: From Oct 7, 1993, to Aug 12, 1999, 335 patients were registered.
Introduction: A single institutional surgical trial for gastric cancer had demonstrated increased morbidity but not mortality. This report analyzes risk factors affecting morbidity.
Methods: Risk factors for morbidity in 221 patients treated with curative intent were evaluated in a prospective randomized trial comparing D1 and D3 surgery for curable gastric cancer.
Background/aims: Having observed a lower survival rate of patients with Borrmann type IV gastric cancer, we attempted to determine its prognostic indicators.
Methodology: A total of 103 patients with Borrmann type IV gastric cancer were evaluated; 604 patients with Borrmann types I, II and III were used as references.
Results: The results showed that Borrmann type IV gastric cancer were larger, had deeper invasion, more lymphatic and vascular invasions, predominant diffuse type and scirrhous stromal reaction, extensive lymph node metastases and peritoneal carcinomatosis.