Tomato -gene-mediated resistance against Fusarium wilt originates in roots and extends to shoots via xylem to limit pathogen colonization.

Vascular wilt disease, caused by the soil-borne fungus (), poses a threat to many crop species. Four different tomato resistance () genes (, , , and ) have been identified to confer protection against f.sp.

The tomato P69 subtilase family is involved in resistance to bacterial wilt.

The intercellular space or apoplast constitutes the main interface in plant-pathogen interactions. Apoplastic subtilisin-like proteases-subtilases-may play an important role in defence and they have been identified as targets of pathogen-secreted effector proteins. Here, we characterise the role of the Solanaceae-specific P69 subtilase family in the interaction between tomato and the vascular bacterial wilt pathogen Ralstonia solanacearum.

Pattern-triggered immunity restricts host colonization by endophytic fusaria, but does not affect endophyte-mediated resistance.

Fusarium oxysporum (Fo) is best known as a host-specific vascular pathogen causing major crop losses. Most Fo strains, however, are root endophytes potentially conferring endophyte-mediated resistance (EMR). EMR is a mechanistically poorly understood root-specific induced resistance response induced by endophytic or nonhost pathogenic Fo strains.

[Evidence of enterotoxins and toxic shock syndrome toxin (TSST-1) in strains of Staphylococcus aureus isolated from various biological materials].

Among 413 strains of S. aureus isolated from patients with chronic staphylococcal infections, from haemocultures in bacteriaemia and septic conditions, from gynaecological materials and nasal plugs of healthy carriers the authors detected the production of one or several types of enterotoxins in 124 strains (30.0%), production of TSST-1 in 19 strains (4.

Significance of specific antibody determination in Lyme borreliosis diagnosis.

The diagnosis of Lyme borreliosis, except in cases characterised by pathognomonic clinical manifestation, usually requires confirmation by means of microbiological diagnostic assay, mainly by antibody detection methods. In our study antibodies to B. burgdorferi were tested in neurological patients with suspected Lyme borreliosis, depending on syndrome and clinical diagnosis.